also reported that the overexpression of C‐X‐C motif chemokine ligand 1 (CXCL1) in mice liver drive steatosis‐to‐nonalcoholic steatohepatitis (NASH) progression through neutrophil‐derived ROS and activation of stress kinases, and this process could be reversed by IL‐22 treatment. The gene discussed is CXCL1; the disease is metabolic dysfunction-associated steatohepatitis.