Furthermore, significantly elevated SKP2:FOXA1 (SKP2 intensity/FOXA1 intensity) was detected in TMA cores at stage IV PCa compared to normal prostate tissue and stages II and III of PCa (Fig. 1E, mean = 1.6, P < 0.0001), suggesting that the SKP2‐FOXA1 interplay increases with PCa severity. The gene discussed is SKP2; the disease is posterior cortical atrophy.