The overexpression of miR-483-3p in endothelial cells inhibits Ang II-induced endothelial dysfunction by targeting several molecules, such as transforming growth factor-beta (TGF-β), connective tissue growth factor (CTGF), angiotensin-converting enzyme 1 (ACE1), and endothelin-1 (ET-1) [73] (Table 2). This evidence concerns the gene EDN1 and endothelial dysfunction.