In addition to GPX4 and SLC7A11, another key regulator of ferroptosis, ACSL4, has been reported to act as a direct downstream target of miR-211-5p in HCC, and miR-211-5p suppresses malignant phenotypes such as cell proliferation, migration, and invasion by inhibiting ACSL4 expression [118]. This evidence concerns the gene GPX4 and hepatocellular carcinoma.