GSK3B and Alzheimer disease: We first confirmed previous data showing aberrant morphological development [34] (namely altered total dendritic length (Supplementary Fig. S4B, D, F, H, J) and number of crossings in Sholl ́s analysis (Supplementary Fig. S4A, C, E, G, and I)) in a mouse model of AD that overexpresses GSK-3β [34], one of the key players in this condition.