Although the reported fusions had different breakpoints inside NUDT3 and RAD51B genes, due to the detection of opposite RAD51::NUDT3 fusion with similar number of fusion crossing reads, we can assume that detected translocation is balanced and reciprocal between chr14 (q24.1) and chr6 (p21.31) in both tumor cases. The gene discussed is RAD51B; the disease is neoplasm.