Quantification of the number of STMN2 CE junctions detected in the excitatory neurons in each of the disease groups showed that there was a significant increase in STMN2 CEs in the C9-FTD group compared to controls in both the frontal cortex (p = 0.00079; Wilcoxon test) and the occipital cortex (p = 0.044; Wilcoxon test), as well as a significant increase in the C9-ALS/FTD group compared to controls in the frontal cortex (p = 0.00048; Wilcoxon test) (Fig. 1e). The gene discussed is STMN2; the disease is frontotemporal dementia.