CX3CR1 and experimental autoimmune encephalomyelitis: More broadly, in the experimental autoimmune encephalomyelitis model of multiple sclerosis, NK cell migration into the CNS is mediated in part by CX3CR1-dependent recruitment [101, 102], suggesting that the differential expression of CX3CR1 in NK cells that we observed in our study could plausibly affect NK cell recruitment to the CNS in primary tauopathy.