In line with our study, impaired dopamine homeostasis remarkably lowered the VDAC1 and VDAC2 levels in human neuroblastoma SH-SY5Y cells [41], suggesting the positive modulation of dopaminergic receptors to VDAC even if the specific dopaminergic subtypes responsible to mediate this process were not clarified. Here, VDAC2 is linked to neuroblastoma.