MFSD2A and metabolic dysfunction-associated steatotic liver disease: Taken together, these findings support the conclusion that Mfsd2a, in parallel with the CDP/choline pathway, is critical for maintaining phospholipids containing 18:2 under HFD challenge, establishing LPC transport via Mfsd2a as a quantitatively and physiologically important pathway by which the liver maintains PC pools and protection from the development of NAFLD.