It played a functional role in epithelial wound healing, as replenishment with recombinant Wnt-5a stimulated diabetic corneal wound healing and expression of putative LESC (K15, K17) and diabetes-suppressed (integrin α3β1 and nidogen-1) markers, alleviating important diabetic changes [3, 17] in these corneas. The gene discussed is KRT15; the disease is diabetes mellitus.