KLF3 and myocardial infarction: Overexpression of lncRNA KLF3-AS1 in hMSC-Exos (human mesenchymal stem cells) significantly improved the cardiac function of rats with MI, inhibited the infiltration of inflammatory cells, pro-inflammatory cytokines IL-18 and IL-1β, and reduced the apoptosis caused by hypoxia and inhibiting lncRNA KLF3-AS1 in hMSC-Exos led to the opposite effect (Mao et al., 2019).