TGFBR2 and myocardial infarction: In mice with MI, ADMSC-Exos upregulated miR-671, reduced TGFBR2 and P-SMad2 at the protein level, alleviated the inflammatory response of mouse myocardial tissue, reduced myocardial fibrosis and inhibited the apoptosis of myocardial tissue and these functions were attenuated in miR-671-inhibited ADMSC-Exos (Wang et al., 2021a).