In a breast cancer gene (BRCA)-deficient TNBC model, olaparib activated the intratumoral cyclic guanosine monophosphate/adenosine monophosphate synthase (cGAS)/stimulator of interferon genes (STING) pathway, stimulated paracrine secretion by dendritic cells, and promoted CD8+ T cell infiltration, activation, and recruitment (Pantelidou et al., 2019). The gene discussed is STING1; the disease is breast cancer.