SOAT1 and ovarian cancer: MSCs are recruited from bone marrow, adipose tissue, and endometrium and are able to differentiate into CAFsMSCs stimulate proliferation, stemness, angiogenesis, and platinum resistanceIL-6 and LIF secreted by MSCs enhance OCSC function in the STAT-dependent wayMSC-derived TGF-β and VEGF/HIF-1α signals contribute to OCSC support and angiogenesisBone marrow MSCs enhance chemoresistance of ovarian cancer by releasing miR-1180 that activates Wnt/β-catenin signaling