Since the seminal identification of the aberrant overexpression and activation of the transcription factor FOXM1 as a key molecular alteration in ovarian cancer, a large body of evidence has documented the pivotal role played by FOXM1 in the malignant phenotypes of ovarian cancer, such as enhanced cell proliferation, survival, invasion, and chemoresistance [4,5]. The gene discussed is FOXM1; the disease is ovarian carcinoma.