Interestingly, genetic forms of PD affect different mitochondria-associated proteins, such as leucine-rich repeat serine/threonine protein kinase 2 (LRRK2), as well as autophagy–lysosomal proteins (e.g., α-Syn; glucosylceramidase beta 1 (GBA1)), trigger mitochondrial alterations, OS, autophagy, and lysosomal dysfunction (e.g., [20,21,22]). Here, GBA1 is linked to Parkinson disease.