The latter included putative loss-of-function PCa mutations previously reported for ARID1A, ATRX, CHD1, CHD3, HDAC4, KMT2A, KMT2D, SETD2, and SMARCA1 [7], with BRMS1, CARM1, EHMT2, GLI3, HDAC1, KDM6B, PRDM16, RBBP5, and REST possessing known roles in PCa [52,53,54,55,56,57,58,59,60]. The gene discussed is KMT2D; the disease is posterior cortical atrophy.