‘Not recovered’ kidneys (consisting of AKI controls and kidneys from all treated groups) had high expression of fibrinogens (FGA, FGB, and FGG), histones (H1s), plasma proteins (TTR, ALB, AMBP, and APOA4), fibrillar collagens, and proteins involved in ECM organization (MATN2, ICAM1, AMBP, TOR1, FBN1, and FN1) as well as uromodulin (Tamm–Horsfall protein). The gene discussed is FBN1; the disease is acute kidney injury.