APOA4 and acute kidney injury: ‘Not recovered’ kidneys (consisting of AKI controls and kidneys from all treated groups) had high expression of fibrinogens (FGA, FGB, and FGG), histones (H1s), plasma proteins (TTR, ALB, AMBP, and APOA4), fibrillar collagens, and proteins involved in ECM organization (MATN2, ICAM1, AMBP, TOR1, FBN1, and FN1) as well as uromodulin (Tamm–Horsfall protein).