Experimental pharmacological research shows that QFY reduced escape latency of AGE-induced AD model rats in the Morris water maze test, decreased the levels of TNF-α, IL-1β, and AGE in the hippocampus, and downregulated the expressions of RAGE and NF-κB in the hippocampus and cortex of rats (13, 14). This evidence concerns the gene IL1B and Alzheimer disease.