TGFB1 and cystic fibrosis: Through the bioinformatics analysis and DLR assay, we first confirmed the direct binding sites between RMST and miR-24-3p; loss- or gain-of-function experiments further showed that miR-24-3p inhibition by antagomiR-24-3p treatment abolished, while agomiR-24-3p reproduced, the RMST knockdown-mediated effects on TGF-β1-induced CF fibrosis.