The HSP70 high levels attenuated the production of inflammatorycytokines by inactivation of NF-κB p65 [25].On the one hand, both in vitro and in vivoresearch have reported that Gln-induced HSP70 triggered the release ofanti-inflammatory cytokines and reduced inflammatory factors via the NF-κB p65pathway, thereby attenuating lung injury after sepsis. Here, NFKB1 is linked to Sepsis.