NLRP3 and myocardial infarction: In vivo, combined exposure to DEHP, BBzP and DIDP at doses comparable to levels experienced post-cardiac surgery activated the NLRP3 inflammasome, increased cardiac dilation and reduced cardiac systolic function, and increased the percentage of pro-inflammatory macrophages in hearts of a murine model of post-myocardial infarction (MI) [33].