Moreover, OIT3 overexpression inhibited the proliferation, migration, and invasion of HCC cells, while HDLBP could bind to lncFAL and stabilize its expression, reducing the vulnerability of cells to ferroptosis.[68] Camptothecin treatment notably decreased HCC cell colony formation and migration, induced HCC cell death, and activated p38MAPK, thereby enhancing ferroptosis in HCC cells.[69] Betula etnensis Raf. This evidence concerns the gene OIT3 and hepatocellular carcinoma.