Overall, the HIF‐1α/PMAN/ELAVL1 regulatory axis promoted GC development and metastasis by inhibiting ferroptosis.[127] STAT3 has been shown to bind to and regulate the expression of consensus DNA response elements in the promoters of FNR‐related genes (GPX4, SLC7A11, and FTH1) to inhibit ferroptosis and induce 5‐FU resistance.[128] Hypoxic conditions in GC promoted the expression of lnc‐CBSLR, which interacted with YTHDF2 and reduced the stability of CBS mRNA by enhancing the binding of YTHDF2 to the m6A site of CBS. Here, STAT3 is linked to gastric cancer.