Consequently, the level of citrulline was lower and I-FABP higher than in the control group (animals without sepsis), reflecting changes in both the number and function of intestinal epithelial cells; citrulline in animals with sepsis decreased as a result of reduced production by damaged enterocytes and I-FABP increased in the bloodstream in the presence of intestinal ischemia. The gene discussed is FABP2; the disease is Sepsis.