NR4A2 and infection: Previous studies have demonstrated the age‐dependent effectiveness of IFN‐λ against infections of human coronavirus, including SARS‐CoV‐2, with low levels of inflammation.[51, 52, 53, 54] Additional research is required to elucidate the mechanisms through which BaP and IFN‐λ regulate the expression of NR4A2, ACE2, and TMPRSS2 in nasal and bronchial epithelial cells.