Previous studies have demonstrated the age‐dependent effectiveness of IFN‐λ against infections of human coronavirus, including SARS‐CoV‐2, with low levels of inflammation.[51, 52, 53, 54] Additional research is required to elucidate the mechanisms through which BaP and IFN‐λ regulate the expression of NR4A2, ACE2, and TMPRSS2 in nasal and bronchial epithelial cells. This evidence concerns the gene TMPRSS2 and infection.