High IP-10 levels on admission and within the first 24h after STEMI were associated with end-systolic volume decrease 12 weeks after STEMI, suggesting that the dynamic biphasic regulation of IP-10 in blood at the acute phase of myocardial infarction possibly confers a cardioprotective role (Figure 5B). Here, CXCL10 is linked to myocardial infarction.