It has been confirmed that myocardial infarction and heart failure can lead to abnormalities in amino acid metabolism such as branched-chain amino acids, taurine or glutamine, and abnormalities in the metabolism of certain amino acids, such as branched-chain amino acids, can activate the mTOR signaling pathway, which accelerates myocardial remodeling and leads to the development of heart failure after myocardial infarction. This evidence concerns the gene MTOR and heart failure.