CD79A and cranioectodermal dysplasia: The mAbs displayed similar levels of Ig mutations as what has previously been reported for TG2‐specific plasma cells in CeD (Figure 2F).[10, 13] Thus, both TG2‐specific and TG3‐specific plasma cells accumulate fewer mutations than other IgA gut plasma cells, indicating that they are generated through equivalent mechanisms, possibly outside of germinal centers.[7] Among the 12 TG3‐specific mAbs, four used IGHV2‐5 in combination with IGKV4‐1 (Table S1, Supporting Information).