We also identified a small disease-specific endothelial cluster that co-expressed cardiomyocyte marker Tnnt2 in the injured mouse heart at 7-day post-myocardial infarction and confirmed its existence in the injured heart tissue using immunofluorescence [45], indicating a fate shift towards cardiomyocyte in response to injuries. Here, TNNT2 is linked to myocardial infarction.