IL17C and neoplasm: In addition, the microflora induces MYD88 in myeloid cells, which triggers IL-23 signaling, accelerates the progression of tumor, and promotes the development of tumor IL-17 responses.[75] And a recent study has shown that the lack of IL-17C can promote the growth and metastasis of lung cancer models.[76] Moreover, certain potential factors also play an important regulatory role in the process of inflammatory infection leading to tumors.