Testis weight (decrease); cryptorchidism and hypospadias (increase); abdominal cryptorchidism (cryptorchidism descends to the opposite side); Testicular testosterone levels (decrease, GD19, PND25); Leydig cell hyperplasia aggregation (early phase, GD17); Abnormal seminiferous cord/tubule formation (SMA decrease, GD19); Sertoli cell‐only (SCO) tubules; Evaluation of Sertoli cell maturation (AMH decrease from GD15‐19 to PND10); SCO tubules (WT‐1); Multinucleated gonocytes were detected in normal cords and in dysgenetic areas (disappeared at PND 10). This evidence concerns the gene AMH and hypospadias.