Notably, NR2F6 KO tumors exhibited significantly greater numbers of progenitor exhaustedCD8+ T cells than WT tumors, an effect not seen in terminally exhausted T cells (Fig. 3F), suggesting that NR2F6 deficiency increases the abundance of functionally intact subset ofexhausted CD8+ T cells that can better control tumor growth. Here, CD8A is linked to neoplasm.