ITGAV and Hepatic fibrosis: Interestingly, individual deletion of β subunit partners of Itgav was unable to effectively inhibit mice liver fibrosis induced by CCl4, indicating a combinatory effect of all the αv integrins to drive fibrosis, which strongly suggests that inhibiting pan‐αv integrins may be required to obtain significant anti‐fibrotic effects.20, 27, 29