Our data consists of samples for one single timepoint, to overcome this limitation, we examined the time-dependent expression of the four potentially clock-regulated glioma drivers APC, HIF1A, TERT and TP53 in HCT116 WT, and in the associated BMAL1, PER2 and NR1D1 KO cells. The gene discussed is BMAL1; the disease is central nervous system cancer.