Yamawaki et al. reported that the use of 2-methylcoprophilinamide (M-COPA) or brefeldin A to inhibit FLT3-ITD’s export from the ER or the use of monensin to inhibit FLT3-ITD’s export from the Golgi apparatus can notably decrease signals of tyrosine phosphorylation in AML [21]. This evidence concerns the gene FLT3 and acute myeloid leukemia.