Here, Oxa reduced protein expression of collagen type 4 alpha 1, galectin, and serpin1, which modulate ECM deposition, as well as moesin, inhibin beta A subunit, and myosin heavy chain 9, which are known to be involved in SLE and LN pathogenesis (Villanueva et al., 2011; Lin et al., 2012; Chen et al., 2014). The gene discussed is MSN; the disease is lobular neoplasia.