POU2AF1 and allergic asthma: Recent studies have shown that IL‐38 could inhibit the activation of intracellular STAT1/3, ERK1/2, P38 MAPK, and NF‐κB signaling pathways, and upregulate the expression of antiallergic response gene RGS13 and host defense‐related gene POU2AF1 in mice with allergic asthma.32