Since type I IFN signaling plays a crucial role in T1D‐related β cell dysfunction, ARGI may be functionally implicated in the pathogenesis of the disease.[2] These results serve as a proof of concept of the potential implication of T1D‐associated lncRNAs in the dysfunction of pancreatic β cells in T1D, and open a new avenue for the development of therapeutic approaches based on lncRNA expression modification. Here, LNCARGI is linked to type 1 diabetes mellitus.