For example, the T1D candidate gene MDA5 is implicated in virus‐induced NFκB activation and pro‐inflammatory gene expression in pancreatic β cells, but it does not participate in viral dsRNA‐induced apoptosis.[6] These results suggest that ARGI might participate in the initial stages of T1D, promoting the inflammatory environment that precedes pancreatic β cell destruction. This evidence concerns the gene LNCARGI and type 1 diabetes mellitus.