Type 1 diabetes (T1D) is a chronic autoimmune disease characterized by the specific destruction of insulin‐producing pancreatic β cells that leads to impaired insulin production and increased blood glucose levels.[1] During the initial stages of the disease, immune cells infiltrate pancreatic islets, generating a pro‐inflammatory environment (insulitis) that is tightly controlled by the release of soluble pro‐inflammatory mediators by both pancreatic β cells and immune cells.[2]. Here, INS is linked to type 1 diabetes mellitus.