Given the small number of samples in our study, however, we cannot distinguish whether the higher general expression of markers (e.g., CD120A, CD123, CD178) in cells derived from T41A mutant tumors thought to be a result of tumor biology with differences in both tumor and stroma resulting from that specific mutation and subsequent signaling alterations, or artifact of the two experiments being performed separately. The gene discussed is FASLG; the disease is neoplasm.