ENAH and infection: This finding indicates that the rSczy3S2-N1038S-CT9△ infection not only has a direct effect on actin binding through Abl2, but also boosts the expression of ENAH, which is inhibited in the other mutated strain, implying that this mutated strain might have an indirect influence on the control of cytoskeletal motion through activating the ENAH pathway with the help of higher ABI1 levels.