On the other hand, the overexpression of hBD3 and LL37, in turn, stimulates keratinocytes to produce pro-inflammatory cytokines (IL-6, interferon gamma-induced protein (IP)-10) and chemokines (MCP-1, MIP3-α, and RANTES) that play an important role in recruiting activated T cells, macrophages, and other immune cells from the peripheral blood into sites of tissue inflammation, which consequently promote proliferation and inflammation in eczema [39,40,41]. This evidence concerns the gene CXCL10 and Eczematoid dermatitis.