Patients with these myopathies have a chance of developing MH that is equivalent to that of the general population, with one exception, represented by hypokalemic periodic paralysis (HypoPP), in most cases caused by mutations in the skeletal muscle voltage-gated Ca2+ channel encoded by CACNA1S (HypoPP type 1) [36]. Here, CACNA1S is linked to hypokalemic periodic paralysis.