NFKB1 and glioblastoma: These studies lack a regulatory network for downstream target genes of NF-κB, therefore, in future research, we need to increase the integrity of the experiment and increase the expression and molecular regulatory networks of downstream target genes of NF-κB signaling to explain the proliferation, migration and invasion, immune escape, or drug resistance of GBM cells, rather than relying on some phenomenon experiments to conclude that this gene regulates GBM cell proliferation, migration and invasion, or immune escape through NF-κB signaling.