The SWI/SNF complex antagonist associated with prostate cancer 1 (SChLAP1, a lncRNA) is increased in primary GBM samples and cell lines and enhances and stabilizes interaction with the protein actinin alpha 4 (ACTN4), which leads to increased nuclear localization of the p65 and activation of NF-κB signaling [75]. This evidence concerns the gene ACTN4 and glioblastoma.