Interestingly, both female and male ME/CFS patients showed similar upregulation of genes related to receptors and proteins that are essential for the development, homeostasis, and survival of immune cells (CXCR2R1, CD200R, CD180, and GIMAPs) and downregulation of genes related to the production of interferons (SCD2 and DDIT3) and metabolism in immune cells (SLCs). This evidence concerns the gene DDIT3 and myalgic encephalomeyelitis/chronic fatigue syndrome.