LIN28B and Hepatic fibrosis: Of note, the presented regulators and interactors were studied in different disease contexts and moreover, even within the same disease occasionally in different models (animals, primary cell culture, cell lines), for which the disease can be induced in different ways, resulting in most cases in the induction of different molecular pathways (for example CCl4 [39,40,41] and alcohol induced liver fibrosis [39], revealing different involved factors in each case e.g., DNMT1 and DNMT3A in the former [39,40,41], Lin28B in the latter [81]).