FUBP1 and central nervous system cancer: Strikingly, more than 80% of the differential splicing events associated with the comparison of FUBP1 LoF vs. FUBP1-WT were exon skipping, which is in agreement with the documented role of FUBP1 in regulating exon splicing and modulating exon inclusion [31,33,34,35], as well as its role in shaping the splicing landscape in low-grade gliomas [31].