HuR was found to stabilize Uqcrb, Ndufb6, and Apob mRNAs; in fact, hepatocyte-specific HuR knockout reduced the expression of the respective protein factors, resulting in a reduction of liver lipid export and ATP production and in the aggravation of HF diet-induced NAFLD [53]. Here, APOB is linked to metabolic dysfunction-associated steatotic liver disease.