The genes were mainly enriched in the GO terms with methyltransferase activity, amino methyltransferase activity, and nucleic acid binding, and in the KEGG pathways with cytokine–cytokine receptor interaction, JAK-STAT signaling pathway, RNA transport, autophagy-other, autophagy-yeast, Fanconi anemia pathway, and nonhomologous end-joining (Supplementary Figure S3). This evidence concerns the gene SOAT1 and Fanconi anemia.