Moreover, Mao et al. [38] reported that the level of CXCL5 was elevated in gastric cancer, which was positively correlated with lymphatic metastasis and tumor differentiation, while recombinant human CXCL5 (rhCXCL5) could induce gastric cancer cell epithelial–mesenchymal transition (EMT) by activating the ERK pathway to enhance the migratory and invasive capacities of tumor cells. The gene discussed is CXCL5; the disease is neoplasm.