In another study, ultraviolet light-induced apoptosis of glioma cell lines is increased by miR-98 treatment through a mechanism dependent on inhibitor of nuclear kappa-B kinase epsilon (IKBKE, also known as IKKi or IKKε), nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB), and Bcl-2 (Figure 5, [72]). The gene discussed is NFKB1; the disease is glioma.